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1.

Evasion of affinity-based selection in germinal centers by Epstein-Barr virus LMP2A.
by Minamitani, Takeharu

Proceedings of the National Academy of Sciences of the United States of America, September 15, 2015, Vol.112(37), pp.11612-11617

2.

Epstein-Barr virus nuclear antigen leader protein localizes to promoters and enhancers with cell transcription factors and EBNA2.
by Portal, Daniel

Proceedings of the National Academy of Sciences of the United States of America, November 12, 2013, Vol.110(46), pp.18537-18542

3.

Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth.
by Zhao, Bo

Proceedings of the National Academy of Sciences of the United States of America, September 6, 2011, Vol.108(36), pp.14902-14907

4.

Nucleolin is important for Epstein-Barr virus nuclear antigen 1-mediated episome binding, maintenance, and transcription.
by Chen, Ya-Lin

Proceedings of the National Academy of Sciences of the United States of America, January 7, 2014, Vol.111(1), pp.243-248

5.

Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A
by Jiang, Sizun

Proceedings of the National Academy of Sciences of the United States of America, 07 January 2014, Vol.111(1), pp.421-6

6.

Epstein-Barr virus nuclear protein EBNA3C residues critical for maintaining lymphoblastoid cell growth.
by Maruo, Seiji

Proceedings of the National Academy of Sciences of the United States of America, March 17, 2009, Vol.106(11), pp.4419-4424

7.

RNAs induced by Epstein–Barr virus nuclear antigen 2 in lymphoblastoid cell lines
by Bo Zhao

Proceedings of the National Academy of Sciences of the United States of America, 07 February 2006, Vol.103(6), p.1900

9.

The X-linked lymphoproliferative syndrome gene product SH2D1A associates with p62dok (Dok1) and activates NF-κB
by Bakary S. Sylla

Proceedings of the National Academy of Sciences of the United States of America, 20 June 2000, Vol.97(13), p.7470

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