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1.

Suppression of Mediator is regulated by Cdk8-dependent Grr1 turnover of the Med3 coactivator
by Gonzalez, Deyarina

Proceedings of the National Academy of Sciences of the United States of America, 18 February 2014, Vol.111(7), pp.2500-5

2.

Structural hierarchy controlling dimerization and target DNA recognition in the AHR transcriptional complex
by Seok, Seung-Hyeon

Proceedings of the National Academy of Sciences of the United States of America, 10 April 2017, Vol.114(21)

3.

Complexity of chromatin folding is captured by the strings and binders switch model.
by Barbieri, Mariano

Proceedings of the National Academy of Sciences of the United States of America, October 2, 2012, Vol.109(40), pp.16173-16178

4.

Aldosterone-stimulating somatic gene mutations are common in normal adrenal glands.
by Nishimoto, Koshiro

Proceedings of the National Academy of Sciences of the United States of America, August 18, 2015, Vol.112(33), pp.E4591-E4599

5.

CDK8/19 Mediator kinases potentiate induction of transcription by NFκB.
by Chen, Mengqian

Proceedings of the National Academy of Sciences of the United States of America, September 19, 2017, Vol.114(38), pp.10208-10213

7.

Period2 3'-UTR and microRNA-24 regulate circadian rhythms by repressing PERIOD2 protein accumulation.
by Yoo, Seung-Hee

Proceedings of the National Academy of Sciences of the United States of America, October 17, 2017, Vol.114(42), pp.E8855-E8864

8.

Mutator activity induced by microRNA-155 (miR-155) links inflammation and cancer.
by Tili, Esmerina

Proceedings of the National Academy of Sciences of the United States of America, March 22, 2011, Vol.108(12), pp.4908-4913

9.

A diversity of paracrine signals sustains molecular circadian cycling in suprachiasmatic nucleus circuits.
by Maywood, Elizabeth S

Proceedings of the National Academy of Sciences of the United States of America, August 23, 2011, Vol.108(34), pp.14306-14311

10.

Critical role for inflammasome-independent IL-1β production in osteomyelitis
by Lukens, John R

Proceedings of the National Academy of Sciences of the United States of America, 21 January 2014, Vol.111(3), pp.1066-71

11.

Direct observation of a transient ternary complex during IκBα-mediated dissociation of NF-κB from DNA
by Alverdi, Vera

Proceedings of the National Academy of Sciences of the United States of America, 07 January 2014, Vol.111(1), pp.225-30

12.

Transcriptional control of amino acid homeostasis is disrupted in Huntington's disease.
by Sbodio, Juan I

Proceedings of the National Academy of Sciences of the United States of America, August 2, 2016, Vol.113(31), pp.8843-8848

14.

Kelch-like 3 and Cullin 3 regulate electrolyte homeostasis via ubiquitination and degradation of WNK4.
by Shibata, Shigeru

Proceedings of the National Academy of Sciences of the United States of America, May 7, 2013, Vol.110(19), pp.7838-7843

15.

Nuclear receptor HNF4A transrepresses CLOCK:BMAL1 and modulates tissue-specific circadian networks.
by Qu, Meng

Proceedings of the National Academy of Sciences of the United States of America, December 26, 2018, Vol.115(52), pp.E12305-E12312

16.

Network-based prediction for sources of transcriptional dysregulation using latent pathway identification analysis.
by Pham, Lisa

Proceedings of the National Academy of Sciences of the United States of America, August 9, 2011, Vol.108(32), pp.13347-13352

17.

NFATc1 and NFATc2 repress spontaneous osteoarthritis.
by Greenblatt, Matthew B

Proceedings of the National Academy of Sciences of the United States of America, December 3, 2013, Vol.110(49), pp.19914-19919

18.

Positional specificity of different transcription factor classes within enhancers.
by Grossman, Sharon R

Proceedings of the National Academy of Sciences of the United States of America, July 24, 2018, Vol.115(30), pp.E7222-E7230

19.

Computational and experimental insights into the circadian effects of SIRT1
by Foteinou, Panagiota T

Proceedings of the National Academy of Sciences of the United States of America, 06 November 2018, Vol.115(45), pp.11643-11648

20.

MTMR3 risk allele enhances innate receptor-induced signaling and cytokines by decreasing autophagy and increasing caspase-1 activation.
by Lahiri, Amit

Proceedings of the National Academy of Sciences of the United States of America, August 18, 2015, Vol.112(33), pp.10461-10466

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