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1.

Suppression of Mediator is regulated by Cdk8-dependent Grr1 turnover of the Med3 coactivator
by Gonzalez, Deyarina

Proceedings of the National Academy of Sciences of the United States of America, 18 February 2014, Vol.111(7), pp.2500-5

2.

Complexity of chromatin folding is captured by the strings and binders switch model.
by Barbieri, Mariano

Proceedings of the National Academy of Sciences of the United States of America, October 2, 2012, Vol.109(40), pp.16173-16178

3.

Aldosterone-stimulating somatic gene mutations are common in normal adrenal glands.
by Nishimoto, Koshiro

Proceedings of the National Academy of Sciences of the United States of America, August 18, 2015, Vol.112(33), pp.E4591-E4599

5.

Period2 3'-UTR and microRNA-24 regulate circadian rhythms by repressing PERIOD2 protein accumulation.
by Yoo, Seung-Hee

Proceedings of the National Academy of Sciences of the United States of America, October 17, 2017, Vol.114(42), pp.E8855-E8864

6.

Mutator activity induced by microRNA-155 (miR-155) links inflammation and cancer.
by Tili, Esmerina

Proceedings of the National Academy of Sciences of the United States of America, March 22, 2011, Vol.108(12), pp.4908-4913

7.

A diversity of paracrine signals sustains molecular circadian cycling in suprachiasmatic nucleus circuits.
by Maywood, Elizabeth S

Proceedings of the National Academy of Sciences of the United States of America, August 23, 2011, Vol.108(34), pp.14306-14311

8.

Silencing synuclein at the synapse with PLK2
by Looyenga, Brendan D

Proceedings of the National Academy of Sciences of the United States of America, 08 October 2013, Vol.110(41), pp.16293-4

9.

Critical role for inflammasome-independent IL-1β production in osteomyelitis
by Lukens, John R

Proceedings of the National Academy of Sciences of the United States of America, 21 January 2014, Vol.111(3), pp.1066-71

10.

Network-based prediction for sources of transcriptional dysregulation using latent pathway identification analysis
by Pham, Lisa

Proceedings of the National Academy of Sciences of the United States of America, 09 August 2011, Vol.108(32), pp.13347-52

11.

NFATc1 and NFATc2 repress spontaneous osteoarthritis
by Greenblatt, Matthew B

Proceedings of the National Academy of Sciences of the United States of America, 03 December 2013, Vol.110(49), pp.19914-9

12.

Direct observation of a transient ternary complex during IκBα-mediated dissociation of NF-κB from DNA
by Alverdi, Vera

Proceedings of the National Academy of Sciences of the United States of America, 07 January 2014, Vol.111(1), pp.225-30

13.

Transcriptional control of amino acid homeostasis is disrupted in Huntington's disease.
by Sbodio, Juan I

Proceedings of the National Academy of Sciences of the United States of America, August 2, 2016, Vol.113(31), pp.8843-8848

15.

Kelch-like 3 and Cullin 3 regulate electrolyte homeostasis via ubiquitination and degradation of WNK4.
by Shibata, Shigeru

Proceedings of the National Academy of Sciences of the United States of America, May 7, 2013, Vol.110(19), pp.7838-7843

16.

Nuclear receptor HNF4A transrepresses CLOCK:BMAL1 and modulates tissue-specific circadian networks.
by Qu, Meng

Proceedings of the National Academy of Sciences of the United States of America, December 26, 2018, Vol.115(52), pp.E12305-E12312

17.

Krüppel-like factor 9 is a circadian transcription factor in human epidermis that controls proliferation of keratinocytes
by Spörl, Florian

Proceedings of the National Academy of Sciences of the United States of America, 03 July 2012, Vol.109(27), pp.10903-8

18.

Circadian clock disruption improves the efficacy of chemotherapy through p73-mediated apoptosis
by Lee, Jin Hyup

Proceedings of the National Academy of Sciences of the United States of America, 28 June 2011, Vol.108(26), pp.10668-72

19.

Computational and experimental insights into the circadian effects of SIRT1
by Foteinou, Panagiota T

Proceedings of the National Academy of Sciences of the United States of America, 06 November 2018, Vol.115(45), pp.11643-11648

20.

MTMR3 risk allele enhances innate receptor-induced signaling and cytokines by decreasing autophagy and increasing caspase-1 activation.
by Lahiri, Amit

Proceedings of the National Academy of Sciences of the United States of America, August 18, 2015, Vol.112(33), pp.10461-10466

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