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1.

Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth.
by Zhao, Bo

Proceedings of the National Academy of Sciences of the United States of America, September 6, 2011, Vol.108(36), pp.14902-14907

2.

Nucleolin is important for Epstein-Barr virus nuclear antigen 1-mediated episome binding, maintenance, and transcription.
by Chen, Ya-Lin

Proceedings of the National Academy of Sciences of the United States of America, January 7, 2014, Vol.111(1), pp.243-248

3.

Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A
by Jiang, Sizun

Proceedings of the National Academy of Sciences of the United States of America, 07 January 2014, Vol.111(1), pp.421-6

4.

RNAs induced by Epstein–Barr virus nuclear antigen 2 in lymphoblastoid cell lines
by Bo Zhao

Proceedings of the National Academy of Sciences of the United States of America, 07 February 2006, Vol.103(6), p.1900

5.

The X-linked lymphoproliferative syndrome gene product SH2D1A associates with p62dok (Dok1) and activates NF-κB
by Bakary S. Sylla

Proceedings of the National Academy of Sciences of the United States of America, 20 June 2000, Vol.97(13), p.7470

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