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1.

Virology. Epstein-Barr virus turns 50.
by Lieberman, Paul M

Science (New York, N.Y.), March 21, 2014, Vol.343(6177), pp.1323-1325

3.

The challenge of diagnosing atypical exanthems: A clinico-laboratory study
by Drago, Francesco

Journal of the American Academy of Dermatology, Dec, 2012, Vol.67(6), p.1282(7)

4.

Nucleolin is important for Epstein-Barr virus nuclear antigen 1-mediated episome binding, maintenance, and transcription.
by Chen, Ya-Lin

Proceedings of the National Academy of Sciences of the United States of America, January 7, 2014, Vol.111(1), pp.243-248

5.

Epstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes.
by Schmidt, Stefanie C S

Proceedings of the National Academy of Sciences of the United States of America, January 13, 2015, Vol.112(2), pp.554-559

9.

Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth.
by Zhao, Bo

Proceedings of the National Academy of Sciences of the United States of America, September 6, 2011, Vol.108(36), pp.14902-14907

10.

Mouse model for acute Epstein-Barr virus infection.
by Wirtz, Tristan

Proceedings of the National Academy of Sciences of the United States of America, November 29, 2016, Vol.113(48), pp.13821-13826

12.

Epstein-Barr virus nuclear antigen leader protein localizes to promoters and enhancers with cell transcription factors and EBNA2.
by Portal, Daniel

Proceedings of the National Academy of Sciences of the United States of America, November 12, 2013, Vol.110(46), pp.18537-18542

14.

Evasion of affinity-based selection in germinal centers by Epstein-Barr virus LMP2A
by Minamitani, Takeharu

Proceedings of the National Academy of Sciences, USA, Sep 2015, Vol.112(37), p.11612

18.

Mouse model of Epstein-Barr virus LMP1- and LMP2A-driven germinal center B-cell lymphoproliferative disease
by Minamitani, Takeharu

Proceedings of the National Academy of Sciences of the United States of America, 02 May 2017, Vol.114(18), pp.4751-4756

19.

Soluble factors produced by activated [CD4.sup.+] T cells modulate EBV latency.(CELL BIOLOGY)(Author abstract)(Report)
by Nagy, Noemi

Proceedings of the National Academy of Sciences of the United States, Jan 31, 2012, Vol.109(5), p.1512(6)

20.

Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A.
by Jiang, Sizun

Proceedings of the National Academy of Sciences of the United States of America, January 7, 2014, Vol.111(1), pp.421-426

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