1.
Androgen Deficiency Exacerbates High-Fat Diet-Induced Metabolic Alterations in Male Mice
by Dubois, Vanessa
Endocrinology (Philadelphia), 2016-02, Vol.157 (2), p.648-665
2.
Cellular Mechanisms by Which FGF21 Improves Insulin Sensitivity in Male Mice
by Camporez, João Paulo G
Endocrinology (Philadelphia), 2013-09, Vol.154 (9), p.3099-3109
3.
Dim Light at Night Exaggerates Weight Gain and Inflammation Associated With a High-Fat Diet in Male Mice
by Fonken, Laura K
Endocrinology (Philadelphia), 2013-10, Vol.154 (10), p.3817-3825
4.
Estrogens Protect against High-Fat Diet-Induced Insulin Resistance and Glucose Intolerance in Mice
by Riant, Elodie
Endocrinology (Philadelphia), 2009-05, Vol.150 (5), p.2109-2117
5.
Metabolism and Energy Expenditure, But Not Feeding or Glucose Tolerance, Are Impaired in Young Kiss1r KO Female Mice
by Tolson, Kristen P
Endocrinology (Philadelphia), 2016-11, Vol.157 (11), p.4192-4199
6.
Deletion of the G Protein-Coupled Receptor 30 Impairs Glucose Tolerance, Reduces Bone Growth, Increases Blood Pressure, and Eliminates Estradiol-Stimulated Insulin Release in Femal...
by Mårtensson, Ulrika E. A
Endocrinology (Philadelphia), 2009-02, Vol.150 (2), p.687-698
7.
Disruption of the Chemokine-Like Receptor-1 (CMKLR1) Gene Is Associated with Reduced Adiposity and Glucose Intolerance
by Ernst, Matthew C
Endocrinology (Philadelphia), 2012-02, Vol.153 (2), p.672-682
8.
Double-Stranded RNA-Activated Protein Kinase Is a Key Modulator of Insulin Sensitivity in Physiological Conditions and in Obesity in Mice
by Carvalho-Filho, M. A
Endocrinology (Philadelphia), 2012-11, Vol.153 (11), p.5261-5274
9.
Gestational Chronodisruption Impairs Circadian Physiology in Rat Male Offspring, Increasing the Risk of Chronic Disease
by Mendez, Natalia
Endocrinology (Philadelphia), 2016-12, Vol.157 (12), p.4654-4668
10.
Chemerin Exacerbates Glucose Intolerance in Mouse Models of Obesity and Diabetes
by Ernst, Matthew C
Endocrinology (Philadelphia), 2010-05, Vol.151 (5), p.1998-2007
11.
A Transient Metabolic Recovery from Early Life Glucose Intolerance in Cystic Fibrosis Ferrets Occurs During Pancreatic Remodeling
by Yi, Yaling
Endocrinology (Philadelphia), 2016-05, Vol.157 (5), p.1852-1865
12.
Transgenerational Inheritance of Glucose Intolerance in a Mouse Model of Neonatal Overnutrition
by Pentinat, Thais
Endocrinology (Philadelphia), 2010-12, Vol.151 (12), p.5617-5623
13.
Combined Deletion of Slc30a7 and Slc30a8 Unmasks a Critical Role for ZnT8 in Glucose-Stimulated Insulin Secretion
by Syring, Kristen E
Endocrinology (Philadelphia), 2016-12, Vol.157 (12), p.4534-4541
14.
Differentially Regulated Protein Kinase A (PKA) Activity in Adipose Tissue and Liver Is Associated With Resistance to Diet-Induced Obesity and Glucose Intolerance in Mice That Lack...
by London, Edra
Endocrinology (Philadelphia), 2014-09, Vol.155 (9), p.3397-3408
15.
Increased Expression of the Glucose-Responsive Gene, RCAN1, Causes Hypoinsulinemia, β-Cell Dysfunction, and Diabetes
by Peiris, Heshan
Endocrinology (Philadelphia), 2012-11, Vol.153 (11), p.5212-5221
16.
Disruption of the Dopamine D2 Receptor Impairs Insulin Secretion and Causes Glucose Intolerance
by García-Tornadú, Isabel
Endocrinology (Philadelphia), 2010-04, Vol.151 (4), p.1441-1450
17.
Blockade of Sphingosine 1-Phosphate Receptor 2 Signaling Attenuates High-Fat Diet-Induced Adipocyte Hypertrophy and Systemic Glucose Intolerance in Mice
by Kitada, Yoshihiko
Endocrinology (Philadelphia), 2016-05, Vol.157 (5), p.1839-1851
18.
Administration of Melatonin and Metformin Prevents Deleterious Effects of Circadian Disruption and Obesity in Male Rats
by Thomas, Anthony P
Endocrinology (Philadelphia), 2016-12, Vol.157 (12), p.4720-4731
19.
Ywhaz/14-3-3ζ Deletion Improves Glucose Tolerance Through a GLP-1-Dependent Mechanism
by Lim, Gareth E
Endocrinology (Philadelphia), 2016-07, Vol.157 (7), p.2649-2659
20.
Brain Glucagon-Like Peptide 1 Signaling Controls the Onset of High-Fat Diet-Induced Insulin Resistance and Reduces Energy Expenditure
by Knauf, Claude
Endocrinology (Philadelphia), 2008-10, Vol.149 (10), p.4768-4777
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