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2.
Renal compartment-specific genetic variation analyses identify new pathways in chronic kidney disease
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Nature medicine, 2018-11, Vol.24 (11), p.1721-1731

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Tissue expression of tubular injury markers is associated with renal function decline in diabetic nephropathy
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Origin and function of myofibroblasts in kidney fibrosis
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Nature medicine, 2013-08, Vol.19 (8), p.1047-1053

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Defective glucose metabolism in polycystic kidney disease identifies a new therapeutic strategy
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Pyruvate kinase M2 activation may protect against the progression of diabetic glomerular pathology and mitochondrial dysfunction
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Nature medicine, 2017-06, Vol.23 (6), p.753-762

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An update on the role of the inflammasomes in the pathogenesis of kidney diseases
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Sodium Depletion Enhances Renal Expression of (Pro)Renin Receptor via Cyclic GMP-Protein Kinase G Signaling Pathway
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Kidney Hypoxia, Attributable to Increased Oxygen Consumption, Induces Nephropathy Independently of Hyperglycemia and Oxidative Stress
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Defective fatty acid oxidation in renal tubular epithelial cells has a key role in kidney fibrosis development
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Nature medicine, 2015-01, Vol.21 (1), p.37-46

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Activin-like kinase 3 is important for kidney regeneration and reversal of fibrosis
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Rapid mass spectrometric conversion of tissue biopsy samples into permanent quantitative digital proteome maps
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Nature medicine, 2015-04, Vol.21 (4), p.407-413

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A comparative study on renal biopsy before and after long-term calcineurin inhibitors therapy: an insight for pathogenesis of its toxicity
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MicroRNA-34a Suppresses Autophagy in Tubular Epithelial Cells in Acute Kidney Injury
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Role of Renal Oxygenation and Mitochondrial Function in the Pathophysiology of Acute Kidney Injury
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Urinary Mitochondrial DNA Copy Number Identifies Chronic Renal Injury in Hypertensive Patients
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Proliferative capacity of stem/progenitor-like cells in the kidney may associate with the outcome of patients with acute tubular necrosis
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