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Acute kidney injury

Summary Acute kidney injury (formerly known as acute renal failure) is a syndrome characterised by the rapid loss of the kidney's excretory function and is typically diagnosed by the accumulation of end products of nitrogen metabolism (urea and creatinine) or decreased urine output, or both. It is t... Full description

Journal Title: The Lancet (British edition) 2012, Vol.380 (9843), p.756-766
Main Author: Bellomo, Rinaldo, Prof
Other Authors: Kellum, John A, MD , Ronco, Claudio, MD
Format: Electronic Article Electronic Article
Language: English
Subjects:
Quelle: Alma/SFX Local Collection
Publisher: Kidlington: Elsevier Ltd
ID: ISSN: 0140-6736
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recordid: cdi_proquest_miscellaneous_1093474386
title: Acute kidney injury
format: Article
creator:
  • Bellomo, Rinaldo, Prof
  • Kellum, John A, MD
  • Ronco, Claudio, MD
subjects:
  • Abridged Index Medicus
  • Acute Kidney Injury - diagnosis
  • Acute Kidney Injury - etiology
  • Acute Kidney Injury - therapy
  • Acute renal failure
  • Biochemistry
  • Bioindicators
  • Biological and medical sciences
  • Biomarkers - blood
  • Care and treatment
  • Chronic kidney failure
  • Clinical Laboratory Techniques - methods
  • Complications
  • Complications and side effects
  • Diagnosis
  • Epidemiology
  • General aspects
  • Hemodialysis
  • Hospitals
  • Humans
  • Injuries
  • Internal Medicine
  • Kidney
  • Kidney diseases
  • Kidneys
  • Medical sciences
  • Mortality
  • Nephrology. Urinary tract diseases
  • Neurotransmitter Agents - physiology
  • Nitrogen
  • Prognosis
  • Renal Replacement Therapy - methods
  • Risk factors
  • Urea
  • Urinary system involvement in other diseases. Miscellaneous
  • Urine
ispartof: The Lancet (British edition), 2012, Vol.380 (9843), p.756-766
description: Summary Acute kidney injury (formerly known as acute renal failure) is a syndrome characterised by the rapid loss of the kidney's excretory function and is typically diagnosed by the accumulation of end products of nitrogen metabolism (urea and creatinine) or decreased urine output, or both. It is the clinical manifestation of several disorders that affect the kidney acutely. Acute kidney injury is common in hospital patients and very common in critically ill patients. In these patients, it is most often secondary to extrarenal events. How such events cause acute kidney injury is controversial. No specific therapies have emerged that can attenuate acute kidney injury or expedite recovery; thus, treatment is supportive. New diagnostic techniques (eg, renal biomarkers) might help with early diagnosis. Patients are given renal replacement therapy if acute kidney injury is severe and biochemical or volume-related, or if uraemic-toxaemia-related complications are of concern. If patients survive their illness and do not have premorbid chronic kidney disease, they typically recover to dialysis independence. However, evidence suggests that patients who have had acute kidney injury are at increased risk of subsequent chronic kidney disease.
language: eng
source: Alma/SFX Local Collection
identifier: ISSN: 0140-6736
fulltext: fulltext
issn:
  • 0140-6736
  • 1474-547X
url: Link


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descriptionSummary Acute kidney injury (formerly known as acute renal failure) is a syndrome characterised by the rapid loss of the kidney's excretory function and is typically diagnosed by the accumulation of end products of nitrogen metabolism (urea and creatinine) or decreased urine output, or both. It is the clinical manifestation of several disorders that affect the kidney acutely. Acute kidney injury is common in hospital patients and very common in critically ill patients. In these patients, it is most often secondary to extrarenal events. How such events cause acute kidney injury is controversial. No specific therapies have emerged that can attenuate acute kidney injury or expedite recovery; thus, treatment is supportive. New diagnostic techniques (eg, renal biomarkers) might help with early diagnosis. Patients are given renal replacement therapy if acute kidney injury is severe and biochemical or volume-related, or if uraemic-toxaemia-related complications are of concern. If patients survive their illness and do not have premorbid chronic kidney disease, they typically recover to dialysis independence. However, evidence suggests that patients who have had acute kidney injury are at increased risk of subsequent chronic kidney disease.
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subjectAbridged Index Medicus ; Acute Kidney Injury - diagnosis ; Acute Kidney Injury - etiology ; Acute Kidney Injury - therapy ; Acute renal failure ; Biochemistry ; Bioindicators ; Biological and medical sciences ; Biomarkers - blood ; Care and treatment ; Chronic kidney failure ; Clinical Laboratory Techniques - methods ; Complications ; Complications and side effects ; Diagnosis ; Epidemiology ; General aspects ; Hemodialysis ; Hospitals ; Humans ; Injuries ; Internal Medicine ; Kidney ; Kidney diseases ; Kidneys ; Medical sciences ; Mortality ; Nephrology. Urinary tract diseases ; Neurotransmitter Agents - physiology ; Nitrogen ; Prognosis ; Renal Replacement Therapy - methods ; Risk factors ; Urea ; Urinary system involvement in other diseases. Miscellaneous ; Urine
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descriptionSummary Acute kidney injury (formerly known as acute renal failure) is a syndrome characterised by the rapid loss of the kidney's excretory function and is typically diagnosed by the accumulation of end products of nitrogen metabolism (urea and creatinine) or decreased urine output, or both. It is the clinical manifestation of several disorders that affect the kidney acutely. Acute kidney injury is common in hospital patients and very common in critically ill patients. In these patients, it is most often secondary to extrarenal events. How such events cause acute kidney injury is controversial. No specific therapies have emerged that can attenuate acute kidney injury or expedite recovery; thus, treatment is supportive. New diagnostic techniques (eg, renal biomarkers) might help with early diagnosis. Patients are given renal replacement therapy if acute kidney injury is severe and biochemical or volume-related, or if uraemic-toxaemia-related complications are of concern. If patients survive their illness and do not have premorbid chronic kidney disease, they typically recover to dialysis independence. However, evidence suggests that patients who have had acute kidney injury are at increased risk of subsequent chronic kidney disease.
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1Acute Kidney Injury - diagnosis
2Acute Kidney Injury - etiology
3Acute Kidney Injury - therapy
4Acute renal failure
5Biochemistry
6Bioindicators
7Biological and medical sciences
8Biomarkers - blood
9Care and treatment
10Chronic kidney failure
11Clinical Laboratory Techniques - methods
12Complications
13Complications and side effects
14Diagnosis
15Epidemiology
16General aspects
17Hemodialysis
18Hospitals
19Humans
20Injuries
21Internal Medicine
22Kidney
23Kidney diseases
24Kidneys
25Medical sciences
26Mortality
27Nephrology. Urinary tract diseases
28Neurotransmitter Agents - physiology
29Nitrogen
30Prognosis
31Renal Replacement Therapy - methods
32Risk factors
33Urea
34Urinary system involvement in other diseases. Miscellaneous
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abstractSummary Acute kidney injury (formerly known as acute renal failure) is a syndrome characterised by the rapid loss of the kidney's excretory function and is typically diagnosed by the accumulation of end products of nitrogen metabolism (urea and creatinine) or decreased urine output, or both. It is the clinical manifestation of several disorders that affect the kidney acutely. Acute kidney injury is common in hospital patients and very common in critically ill patients. In these patients, it is most often secondary to extrarenal events. How such events cause acute kidney injury is controversial. No specific therapies have emerged that can attenuate acute kidney injury or expedite recovery; thus, treatment is supportive. New diagnostic techniques (eg, renal biomarkers) might help with early diagnosis. Patients are given renal replacement therapy if acute kidney injury is severe and biochemical or volume-related, or if uraemic-toxaemia-related complications are of concern. If patients survive their illness and do not have premorbid chronic kidney disease, they typically recover to dialysis independence. However, evidence suggests that patients who have had acute kidney injury are at increased risk of subsequent chronic kidney disease.
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pubElsevier Ltd
pmid22617274
doi10.1016/S0140-6736(11)61454-2