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Angiotensin II receptor antagonist reduces urinary liver-type fatty acid-binding protein levels in patients with diabetic nephropathy and chronic renal failure

Byline: T. Nakamura (1), T. Sugaya (2), H. Koide (3) Author Affiliation: (1) Department of Medicine, Shinmatsudo Central General Hospital, Chiba, Japan (2) Research Unit for Organ Regeneration, Riken Kobe Institute, Hyogo, Japan (3) Department of Medicine, Koto Hospital, 6-8-5 Ojima, Koto-ku, Tokyo,... Full description

Journal Title: Diabetologia 2007, Vol.50 (2), p.490-492
Main Author: NAKAMURA, T
Other Authors: SUGAYA, T , KOIDE, H
Format: Electronic Article Electronic Article
Language: English
Subjects:
Angiotensin
Angiotensin Receptor Antagonists
Angiotensin-Converting Enzyme Inhibitors - therapeutic use
Associated diseases and complications
Benzimidazoles - therapeutic use
Benzoates - therapeutic use
Biological and medical sciences
Care and treatment
Chronic kidney failure
Creatinine - blood
Diabetes. Impaired glucose tolerance
Diabetic nephropathies
Diabetic Nephropathies - drug therapy
Diabetic Nephropathies - urine
Endocrine pancreas. Apud cells (diseases)
Endocrinopathies
Fatty Acid-Binding Proteins - urine
Fatty acids
Gastroenterology. Liver. Pancreas. Abdomen
Glomerular Filtration Rate
Humans
Kidney Failure, Chronic - drug therapy
Kidney Failure, Chronic - prevention & control
Kidney Failure, Chronic - urine
Kidneys
Liver. Biliary tract. Portal circulation. Exocrine pancreas
Medical sciences
Nephrology. Urinary tract diseases
Nephropathies. Renovascular diseases. Renal failure
Other diseases. Semiology
Protein binding
Proteins
Renal failure
Urinary system involvement in other diseases. Miscellaneous
Publisher: Berlin: Springer
ID: ISSN: 0012-186X
Zum Text:
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title: Angiotensin II receptor antagonist reduces urinary liver-type fatty acid-binding protein levels in patients with diabetic nephropathy and chronic renal failure
format: Article
creator:
  • NAKAMURA, T
  • SUGAYA, T
  • KOIDE, H
subjects:
  • Angiotensin
  • Angiotensin Receptor Antagonists
  • Angiotensin-Converting Enzyme Inhibitors - therapeutic use
  • Associated diseases and complications
  • Benzimidazoles - therapeutic use
  • Benzoates - therapeutic use
  • Biological and medical sciences
  • Care and treatment
  • Chronic kidney failure
  • Creatinine - blood
  • Diabetes. Impaired glucose tolerance
  • Diabetic nephropathies
  • Diabetic Nephropathies - drug therapy
  • Diabetic Nephropathies - urine
  • Endocrine pancreas. Apud cells (diseases)
  • Endocrinopathies
  • Fatty Acid-Binding Proteins - urine
  • Fatty acids
  • Gastroenterology. Liver. Pancreas. Abdomen
  • Glomerular Filtration Rate
  • Humans
  • Kidney Failure, Chronic - drug therapy
  • Kidney Failure, Chronic - prevention & control
  • Kidney Failure, Chronic - urine
  • Kidneys
  • Liver. Biliary tract. Portal circulation. Exocrine pancreas
  • Medical sciences
  • Nephrology. Urinary tract diseases
  • Nephropathies. Renovascular diseases. Renal failure
  • Other diseases. Semiology
  • Protein binding
  • Proteins
  • Renal failure
  • Urinary system involvement in other diseases. Miscellaneous
ispartof: Diabetologia, 2007, Vol.50 (2), p.490-492
description: Byline: T. Nakamura (1), T. Sugaya (2), H. Koide (3) Author Affiliation: (1) Department of Medicine, Shinmatsudo Central General Hospital, Chiba, Japan (2) Research Unit for Organ Regeneration, Riken Kobe Institute, Hyogo, Japan (3) Department of Medicine, Koto Hospital, 6-8-5 Ojima, Koto-ku, Tokyo, 136-0072, Japan Article History: Registration Date: 13/11/2006 Received Date: 25/07/2006 Accepted Date: 01/11/2006 Online Date: 15/12/2006
language: eng
source:
identifier: ISSN: 0012-186X
fulltext: no_fulltext
issn:
  • 0012-186X
  • 1432-0428
url: Link


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titleAngiotensin II receptor antagonist reduces urinary liver-type fatty acid-binding protein levels in patients with diabetic nephropathy and chronic renal failure
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descriptionByline: T. Nakamura (1), T. Sugaya (2), H. Koide (3) Author Affiliation: (1) Department of Medicine, Shinmatsudo Central General Hospital, Chiba, Japan (2) Research Unit for Organ Regeneration, Riken Kobe Institute, Hyogo, Japan (3) Department of Medicine, Koto Hospital, 6-8-5 Ojima, Koto-ku, Tokyo, 136-0072, Japan Article History: Registration Date: 13/11/2006 Received Date: 25/07/2006 Accepted Date: 01/11/2006 Online Date: 15/12/2006
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languageeng
publisherBerlin: Springer
subjectAngiotensin ; Angiotensin Receptor Antagonists ; Angiotensin-Converting Enzyme Inhibitors - therapeutic use ; Associated diseases and complications ; Benzimidazoles - therapeutic use ; Benzoates - therapeutic use ; Biological and medical sciences ; Care and treatment ; Chronic kidney failure ; Creatinine - blood ; Diabetes. Impaired glucose tolerance ; Diabetic nephropathies ; Diabetic Nephropathies - drug therapy ; Diabetic Nephropathies - urine ; Endocrine pancreas. Apud cells (diseases) ; Endocrinopathies ; Fatty Acid-Binding Proteins - urine ; Fatty acids ; Gastroenterology. Liver. Pancreas. Abdomen ; Glomerular Filtration Rate ; Humans ; Kidney Failure, Chronic - drug therapy ; Kidney Failure, Chronic - prevention & control ; Kidney Failure, Chronic - urine ; Kidneys ; Liver. Biliary tract. Portal circulation. Exocrine pancreas ; Medical sciences ; Nephrology. Urinary tract diseases ; Nephropathies. Renovascular diseases. Renal failure ; Other diseases. Semiology ; Protein binding ; Proteins ; Renal failure ; Urinary system involvement in other diseases. Miscellaneous
ispartofDiabetologia, 2007, Vol.50 (2), p.490-492
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0Angiotensin II receptor antagonist reduces urinary liver-type fatty acid-binding protein levels in patients with diabetic nephropathy and chronic renal failure
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descriptionByline: T. Nakamura (1), T. Sugaya (2), H. Koide (3) Author Affiliation: (1) Department of Medicine, Shinmatsudo Central General Hospital, Chiba, Japan (2) Research Unit for Organ Regeneration, Riken Kobe Institute, Hyogo, Japan (3) Department of Medicine, Koto Hospital, 6-8-5 Ojima, Koto-ku, Tokyo, 136-0072, Japan Article History: Registration Date: 13/11/2006 Received Date: 25/07/2006 Accepted Date: 01/11/2006 Online Date: 15/12/2006
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0Angiotensin
1Angiotensin Receptor Antagonists
2Angiotensin-Converting Enzyme Inhibitors - therapeutic use
3Associated diseases and complications
4Benzimidazoles - therapeutic use
5Benzoates - therapeutic use
6Biological and medical sciences
7Care and treatment
8Chronic kidney failure
9Creatinine - blood
10Diabetes. Impaired glucose tolerance
11Diabetic nephropathies
12Diabetic Nephropathies - drug therapy
13Diabetic Nephropathies - urine
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15Endocrinopathies
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18Gastroenterology. Liver. Pancreas. Abdomen
19Glomerular Filtration Rate
20Humans
21Kidney Failure, Chronic - drug therapy
22Kidney Failure, Chronic - prevention & control
23Kidney Failure, Chronic - urine
24Kidneys
25Liver. Biliary tract. Portal circulation. Exocrine pancreas
26Medical sciences
27Nephrology. Urinary tract diseases
28Nephropathies. Renovascular diseases. Renal failure
29Other diseases. Semiology
30Protein binding
31Proteins
32Renal failure
33Urinary system involvement in other diseases. Miscellaneous
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1Angiotensin Receptor Antagonists
2Angiotensin-Converting Enzyme Inhibitors - therapeutic use
3Associated diseases and complications
4Benzimidazoles - therapeutic use
5Benzoates - therapeutic use
6Biological and medical sciences
7Care and treatment
8Chronic kidney failure
9Creatinine - blood
10Diabetes. Impaired glucose tolerance
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13Diabetic Nephropathies - urine
14Endocrine pancreas. Apud cells (diseases)
15Endocrinopathies
16Fatty Acid-Binding Proteins - urine
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18Gastroenterology. Liver. Pancreas. Abdomen
19Glomerular Filtration Rate
20Humans
21Kidney Failure, Chronic - drug therapy
22Kidney Failure, Chronic - prevention & control
23Kidney Failure, Chronic - urine
24Kidneys
25Liver. Biliary tract. Portal circulation. Exocrine pancreas
26Medical sciences
27Nephrology. Urinary tract diseases
28Nephropathies. Renovascular diseases. Renal failure
29Other diseases. Semiology
30Protein binding
31Proteins
32Renal failure
33Urinary system involvement in other diseases. Miscellaneous
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abstractByline: T. Nakamura (1), T. Sugaya (2), H. Koide (3) Author Affiliation: (1) Department of Medicine, Shinmatsudo Central General Hospital, Chiba, Japan (2) Research Unit for Organ Regeneration, Riken Kobe Institute, Hyogo, Japan (3) Department of Medicine, Koto Hospital, 6-8-5 Ojima, Koto-ku, Tokyo, 136-0072, Japan Article History: Registration Date: 13/11/2006 Received Date: 25/07/2006 Accepted Date: 01/11/2006 Online Date: 15/12/2006
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