schliessen

Filtern

 

Bibliotheken

Baicalin Attenuates Cardiac Dysfunction and Myocardial Remodeling in a Chronic Pressure-Overload Mice Model

Background/Aims: Baicalin has been shown to be effective for various animal models of cardiovascular diseases, such as pulmonary hypertension, atherosclerosis and myocardial ischaemic injury. However, whether baicalin plays a role in cardiac hypertrophy remains unknown. Here we investigated the prot... Full description

Journal Title: Cellular Physiology and Biochemistry May 2017, Vol.41(3), pp.849-864
Main Author: Zhang, Yanqing
Other Authors: Liao, Pingping , Li, Wei , Hu, Dan , Guan, Siming , Long, Chen
Format: Electronic Article Electronic Article
Language: English
Subjects:
ID: ISSN: 10158987 ; E-ISSN: 14219778
Link: http://search.proquest.com/docview/1990815536/?pq-origsite=primo
Zum Text:
SendSend as email Add to Book BagAdd to Book Bag
Staff View
recordid: proquest1990815536
title: Baicalin Attenuates Cardiac Dysfunction and Myocardial Remodeling in a Chronic Pressure-Overload Mice Model
format: Article
creator:
  • Zhang, Yanqing
  • Liao, Pingping
  • Li, Wei
  • Hu, Dan
  • Guan, Siming
  • Long, Chen
subjects:
  • Beijing China
  • China
  • Homeostasis
  • Gene Expression
  • Heart Attacks
  • Cardiomyocytes
  • Heart Failure
  • Hypertension
  • Hypoxia
  • Fatty Acids
  • Medical Research
  • Ostomy
  • Cardiomyopathy
  • Metabolism
  • Lipids
  • Apoptosis
  • Kinases
  • Rodents
  • Laboratory Animals
  • Hypertrophy
ispartof: Cellular Physiology and Biochemistry, May 2017, Vol.41(3), pp.849-864
description: Background/Aims: Baicalin has been shown to be effective for various animal models of cardiovascular diseases, such as pulmonary hypertension, atherosclerosis and myocardial ischaemic injury. However, whether baicalin plays a role in cardiac hypertrophy remains unknown. Here we investigated the protective...
language: eng
source:
identifier: ISSN: 10158987 ; E-ISSN: 14219778
fulltext: fulltext
issn:
  • 10158987
  • 1015-8987
  • 14219778
  • 1421-9778
url: Link


@attributes
ID817779115
RANK0.07
NO1
SEARCH_ENGINEprimo_central_multiple_fe
SEARCH_ENGINE_TYPEPrimo Central Search Engine
LOCALfalse
PrimoNMBib
record
control
sourcerecordid1990815536
sourceidproquest
recordidTN_proquest1990815536
sourcesystemPC
pqid1990815536
display
typearticle
titleBaicalin Attenuates Cardiac Dysfunction and Myocardial Remodeling in a Chronic Pressure-Overload Mice Model
creatorZhang, Yanqing ; Liao, Pingping ; Li, Wei ; Hu, Dan ; Guan, Siming ; Long, Chen
ispartofCellular Physiology and Biochemistry, May 2017, Vol.41(3), pp.849-864
identifierISSN: 10158987 ; E-ISSN: 14219778
subjectBeijing China ; China ; Homeostasis ; Gene Expression ; Heart Attacks ; Cardiomyocytes ; Heart Failure ; Hypertension ; Hypoxia ; Fatty Acids ; Medical Research ; Ostomy ; Cardiomyopathy ; Metabolism ; Lipids ; Apoptosis ; Kinases ; Rodents ; Laboratory Animals ; Hypertrophy
descriptionBackground/Aims: Baicalin has been shown to be effective for various animal models of cardiovascular diseases, such as pulmonary hypertension, atherosclerosis and myocardial ischaemic injury. However, whether baicalin plays a role in cardiac hypertrophy remains unknown. Here we investigated the protective...
languageeng
source
version5
lds50peer_reviewed
links
openurl$$Topenurl_article
openurlfulltext$$Topenurlfull_article
backlink$$Uhttp://search.proquest.com/docview/1990815536/?pq-origsite=primo$$EView_record_in_ProQuest_(subscribers_only)
search
creatorcontrib
0Zhang, Yanqing
1Liao, Pingping
2Li, Wei
3Hu, Dan
4Guan, Siming
5Long, Chen
titleBaicalin Attenuates Cardiac Dysfunction and Myocardial Remodeling in a Chronic Pressure-Overload Mice Model
descriptionBackground/Aims: Baicalin has been shown to be effective for various animal models of cardiovascular diseases, such as pulmonary hypertension, atherosclerosis and myocardial ischaemic injury. However, whether baicalin plays a role in cardiac hypertrophy remains unknown. Here we investigated the protective...
subject
0Beijing China
1China
2Homeostasis
3Gene Expression
4Heart Attacks
5Cardiomyocytes
6Heart Failure
7Hypertension
8Hypoxia
9Fatty Acids
10Medical Research
11Ostomy
12Cardiomyopathy
13Metabolism
14Lipids
15Apoptosis
16Kinases
17Rodents
18Laboratory Animals
19Hypertrophy
20Pressure overload
21Baicalin
general
0English
1S. Karger AG
2Medical Database
3Health & Medical Collection (Alumni edition)
4Medical Database (Alumni edition)
5Health & Medical Collection
6ProQuest Central
7ProQuest Hospital Collection
8Hospital Premium Collection (Alumni edition)
9ProQuest Health & Medical Complete
10ProQuest Medical Library
11ProQuest Central (new)
12Health Research Premium Collection
13Health Research Premium Collection (Alumni edition)
14ProQuest Central China
15ProQuest One Academic
sourceidproquest
recordidproquest1990815536
issn
010158987
11015-8987
214219778
31421-9778
rsrctypearticle
creationdate2017
addtitleCellular Physiology and Biochemistry
searchscope
01000273
11006761
21006762
31007067
41008875
51008886
61009127
71009386
81009698
910000039
1010000047
1110000118
1210000119
1310000155
1410000156
1510000157
1610000158
1710000255
1810000256
1910000270
2010000271
2110000300
2210000302
2310000347
2410000348
2510000360
26proquest
scope
01000273
11006761
21006762
31007067
41008875
51008886
61009127
71009386
81009698
910000039
1010000047
1110000118
1210000119
1310000155
1410000156
1510000157
1610000158
1710000255
1810000256
1910000270
2010000271
2110000300
2210000302
2310000347
2410000348
2510000360
26proquest
lsr43
01000273true
11006761true
21006762true
31007067true
41008875true
51008886true
61009127true
71009386true
81009698true
910000039true
1010000047true
1110000118true
1210000119true
1310000155true
1410000156true
1510000157true
1610000158true
1710000255true
1810000256true
1910000270true
2010000271true
2110000300true
2210000302true
2310000347true
2410000348true
2510000360true
startdate20170501
enddate20170501
citationpf 849 pt 864 vol 41 issue 3
lsr30VSR-Enriched:[pqid, doi]
sort
titleBaicalin Attenuates Cardiac Dysfunction and Myocardial Remodeling in a Chronic Pressure-Overload Mice Model
authorZhang, Yanqing ; Liao, Pingping ; Li, Wei ; Hu, Dan ; Guan, Siming ; Long, Chen
creationdate20170501
lso0120170501
facets
frbrgroupid8133151418798181284
frbrtype5
newrecords20180131
languageeng
creationdate2017
topic
0Beijing China
1China
2Homeostasis
3Gene Expression
4Heart Attacks
5Cardiomyocytes
6Heart Failure
7Hypertension
8Hypoxia
9Fatty Acids
10Medical Research
11Ostomy
12Cardiomyopathy
13Metabolism
14Lipids
15Apoptosis
16Kinases
17Rodents
18Laboratory Animals
19Hypertrophy
collection
0Medical Database
1Health & Medical Collection (Alumni edition)
2Medical Database (Alumni edition)
3Health & Medical Collection
4ProQuest Central
5ProQuest Hospital Collection
6Hospital Premium Collection (Alumni edition)
7ProQuest Health & Medical Complete
8ProQuest Medical Library
9ProQuest Central (new)
10Health Research Premium Collection
11Health Research Premium Collection (Alumni edition)
12ProQuest Central China
13ProQuest One Academic
prefilterarticles
rsrctypearticles
creatorcontrib
0Zhang, Yanqing
1Liao, Pingping
2Li, Wei
3Hu, Dan
4Guan, Siming
5Long, Chen
jtitleCellular Physiology and Biochemistry
toplevelpeer_reviewed
delivery
delcategoryRemote Search Resource
fulltextfulltext
addata
aulast
0Zhang
1Liao
2Li
3Hu
4Guan
5Long
aufirst
0Yanqing
1Pingping
2Wei
3Dan
4Siming
5Chen
au
0Zhang, Yanqing
1Liao, Pingping
2Li, Wei
3Hu, Dan
4Guan, Siming
5Long, Chen
atitleBaicalin Attenuates Cardiac Dysfunction and Myocardial Remodeling in a Chronic Pressure-Overload Mice Model
jtitleCellular Physiology and Biochemistry
risdate20170501
volume41
issue3
spage849
epage864
pages849-864
issn10158987
eissn14219778
formatjournal
genrearticle
ristypeJOUR
abstractBackground/Aims: Baicalin has been shown to be effective for various animal models of cardiovascular diseases, such as pulmonary hypertension, atherosclerosis and myocardial ischaemic injury. However, whether baicalin plays a role in cardiac hypertrophy remains unknown. Here we investigated the protective...
copBasel
pubS. Karger AG
urlhttp://search.proquest.com/docview/1990815536/
doi10.1159/000459708
date2017-05-01