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Reduction in activating transcription factor 4 promotes carbon tetrachloride and lipopolysaccharide/D-galactosamine-mediated liver injury in mice

Although activating transcription factor 4 (ATF4) is involved in the regulation of numerous biological functions, whether ATF4 has a direct role in liver injury is unknown. The aim of the present study was to investigate the role of ATF4 in liver injury using mouse models. The results revealed that... Full description

Journal Title: Molecular Medicine Reports 2018, Vol.18(2), pp.1718-1725
Main Author: Zhao, Xiaofang
Other Authors: Zhou, Hong , Cheng, Ying , Yu, Wenjing , Luo, Guosong , Duan, Chunyan , Yao, Fuli , Xiao, Bin , Feng, Chunhong , Xia, Xianming , Wei, Mei , Wang, Yong , Li, Jing , Dai, Rongyang
Format: Electronic Article Electronic Article
Language: English
Subjects:
ID: ISSN: 17912997 ; E-ISSN: 17913004 ; DOI: 10.3892/mmr.2018.9080
Link: http://search.proquest.com/docview/2096261123/?pq-origsite=primo
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title: Reduction in activating transcription factor 4 promotes carbon tetrachloride and lipopolysaccharide/D-galactosamine-mediated liver injury in mice
format: Article
creator:
  • Zhao, Xiaofang
  • Zhou, Hong
  • Cheng, Ying
  • Yu, Wenjing
  • Luo, Guosong
  • Duan, Chunyan
  • Yao, Fuli
  • Xiao, Bin
  • Feng, Chunhong
  • Xia, Xianming
  • Wei, Mei
  • Wang, Yong
  • Li, Jing
  • Dai, Rongyang
subjects:
  • United States–Us
  • Germany
  • China
  • Investigations
  • Phosphorylation
  • Endoplasmic Reticulum
  • Alanine Transaminase
  • Crispr
  • Activating Transcription Factor 4
  • Jnk Protein
  • Metabolism
  • Animal Models
  • Liver Diseases
  • Proteins
  • Statistical Analysis
  • Plasmids
  • Alanine
  • Crispr
  • Glycerol
ispartof: Molecular Medicine Reports, 2018, Vol.18(2), pp.1718-1725
description: Although activating transcription factor 4 (ATF4) is involved in the regulation of numerous biological functions, whether ATF4 has a direct role in liver injury is unknown. The aim of the present study was to investigate the role of ATF4 in liver injury using mouse models. The results revealed that ATF4...
language: eng
source:
identifier: ISSN: 17912997 ; E-ISSN: 17913004 ; DOI: 10.3892/mmr.2018.9080
fulltext: fulltext
issn:
  • 17912997
  • 1791-2997
  • 17913004
  • 1791-3004
url: Link


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titleReduction in activating transcription factor 4 promotes carbon tetrachloride and lipopolysaccharide/D-galactosamine-mediated liver injury in mice
creatorZhao, Xiaofang ; Zhou, Hong ; Cheng, Ying ; Yu, Wenjing ; Luo, Guosong ; Duan, Chunyan ; Yao, Fuli ; Xiao, Bin ; Feng, Chunhong ; Xia, Xianming ; Wei, Mei ; Wang, Yong ; Li, Jing ; Dai, Rongyang
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subjectUnited States–Us ; Germany ; China ; Investigations ; Phosphorylation ; Endoplasmic Reticulum ; Alanine Transaminase ; Crispr ; Activating Transcription Factor 4 ; Jnk Protein ; Metabolism ; Animal Models ; Liver Diseases ; Proteins ; Statistical Analysis ; Plasmids ; Alanine ; Crispr ; Glycerol
descriptionAlthough activating transcription factor 4 (ATF4) is involved in the regulation of numerous biological functions, whether ATF4 has a direct role in liver injury is unknown. The aim of the present study was to investigate the role of ATF4 in liver injury using mouse models. The results revealed that ATF4...
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titleReduction in activating transcription factor 4 promotes carbon tetrachloride and lipopolysaccharide/D-galactosamine-mediated liver injury in mice
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titleReduction in activating transcription factor 4 promotes carbon tetrachloride and lipopolysaccharide/D-galactosamine-mediated liver injury in mice
authorZhao, Xiaofang ; Zhou, Hong ; Cheng, Ying ; Yu, Wenjing ; Luo, Guosong ; Duan, Chunyan ; Yao, Fuli ; Xiao, Bin ; Feng, Chunhong ; Xia, Xianming ; Wei, Mei ; Wang, Yong ; Li, Jing ; Dai, Rongyang
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abstractAlthough activating transcription factor 4 (ATF4) is involved in the regulation of numerous biological functions, whether ATF4 has a direct role in liver injury is unknown. The aim of the present study was to investigate the role of ATF4 in liver injury using mouse models. The results revealed that ATF4...
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urlhttp://search.proquest.com/docview/2096261123/
pages1718-1725
date2018-01-01