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Benefits of catalase overexpression in renal proximal tubular cells

The intrarenal renin-angiotensin system and reactive oxygen species contribute to the progression of chronic kidney disease. Godin et al. applied transgenic methods to prove renoprotection by catalase. They show that all of the pathological effects of overexpression of angiotensinogen can be attribu... Full description

Journal Title: Kidney International Jun 2010, Vol.77(12), pp.1060-2
Main Author: Satoh, Minoru
Format: Electronic Article Electronic Article
Language: English
Subjects:
ID: ISSN: 00852538 ; DOI: 10.1038/ki.2010.94
Link: http://search.proquest.com/docview/347368916/?pq-origsite=primo
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title: Benefits of catalase overexpression in renal proximal tubular cells
format: Article
creator:
  • Satoh, Minoru
subjects:
  • Angiotensinogen–Administration & Dosage
  • Animals–Genetics
  • Catalase–Therapeutic Use
  • Catalase–Methods
  • Catalase–Metabolism
  • Disease Progression–Cytology
  • Gene Expression–Drug Effects
  • Genetic Therapy–Drug Effects
  • Humans–Drug Effects
  • Hydrogen Peroxide–Drug Effects
  • Kidney Tubules, Proximal–Drug Effects
  • Kidney Tubules, Proximal–Drug Effects
  • Mice–Drug Effects
  • Reactive Oxygen Species–Drug Effects
  • Renin-Angiotensin System–Drug Effects
  • Reactive Oxygen Species
  • Angiotensinogen
  • Hydrogen Peroxide
  • Catalase
ispartof: Kidney International, Jun 2010, Vol.77(12), pp.1060-2
description: The intrarenal renin-angiotensin system and reactive oxygen species contribute to the progression of chronic kidney disease. Godin et al. applied transgenic methods to prove renoprotection by catalase. They show that all of the pathological effects of overexpression of angiotensinogen can be attributed to stimulation of hydrogen peroxide pathways. This indicates a protective role for catalase and underscores the importance of the intrarenal renin-angiotensin system in renal disease.
language: eng
source:
identifier: ISSN: 00852538 ; DOI: 10.1038/ki.2010.94
fulltext: fulltext
issn:
  • 00852538
  • 0085-2538
url: Link


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identifierISSN: 00852538 ; DOI: 10.1038/ki.2010.94
subjectAngiotensinogen–Administration & Dosage ; Animals–Genetics ; Catalase–Therapeutic Use ; Catalase–Methods ; Catalase–Metabolism ; Disease Progression–Cytology ; Gene Expression–Drug Effects ; Genetic Therapy–Drug Effects ; Humans–Drug Effects ; Hydrogen Peroxide–Drug Effects ; Kidney Tubules, Proximal–Drug Effects ; Kidney Tubules, Proximal–Drug Effects ; Mice–Drug Effects ; Reactive Oxygen Species–Drug Effects ; Renin-Angiotensin System–Drug Effects ; Reactive Oxygen Species ; Angiotensinogen ; Hydrogen Peroxide ; Catalase
descriptionThe intrarenal renin-angiotensin system and reactive oxygen species contribute to the progression of chronic kidney disease. Godin et al. applied transgenic methods to prove renoprotection by catalase. They show that all of the pathological effects of overexpression of angiotensinogen can be attributed to stimulation of hydrogen peroxide pathways. This indicates a protective role for catalase and underscores the importance of the intrarenal renin-angiotensin system in renal disease.
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titleBenefits of catalase overexpression in renal proximal tubular cells
descriptionThe intrarenal renin-angiotensin system and reactive oxygen species contribute to the progression of chronic kidney disease. Godin et al. applied transgenic methods to prove renoprotection by catalase. They show that all of the pathological effects of overexpression of angiotensinogen can be attributed to stimulation of hydrogen peroxide pathways. This indicates a protective role for catalase and underscores the importance of the intrarenal renin-angiotensin system in renal disease.
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abstractThe intrarenal renin-angiotensin system and reactive oxygen species contribute to the progression of chronic kidney disease. Godin et al. applied transgenic methods to prove renoprotection by catalase. They show that all of the pathological effects of overexpression of angiotensinogen can be attributed to stimulation of hydrogen peroxide pathways. This indicates a protective role for catalase and underscores the importance of the intrarenal renin-angiotensin system in renal disease.
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doi10.1038/ki.2010.94
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date2010-06-01